Sato, A., Kishida, S., Tanaka, T., Kikuchi, A., Kodama, T., Asashima,, M., and Nishinakamura, R. Sall1, a causative gene for Townes-Brocks syndrome, enhances the canonical Wnt signaling by localizing to heterochromatin. Biochem. Biophys. Res. Commun, in press.

Takasato, M., Osafune, K., Matsumoto, Y., Kataoka, Y., Yoshida, N., Meguro, H., Aburatani, H., Asashima, M., Nishinakamura, R. Identification of kidney mesenchymal genes by a combination of microarray analysis and Sall1-GFP knockin mice. Mech Dev. 121 (6):547-557, 2004

Nakayama N, Han CY, Cam L, Lee JI, Pretorius J, Fisher S, Rosenfeld R, Scully S, Nishinakamura R, Duryea D, Van G, Bolon B, Yokota T, Zhang K. A novel chordin-like BMP inhibitor, CHL2, expressed preferentially in chondrocytes of developing cartilage and osteoarthritic joint cartilage. Development. 2004; 131(1): 229-240.

Nishinakamura, R. Kidney development conserverd overspecies:essential roles of Sall1. Semin. Cell Dev. Biol. 14 (4):241-247, 2003

Sato, A., Matsumoto, Y., Koide, U., Kataoka, Y., Yoshida, N,. Yokota, T., Asashima, M., and Nishinakamura, R. Zinc finger protein Sall2 is not essential for embryonic and kidney development. Mol. Cell. Biol. 23 (1):62-69, 2003

Osafune, K., Nishinakamura, R., Komazaki, S., and Asashima, M. In vitro induction of the pronephric duct in Xenopus explants. Dev. Growth. Differ. 44 (2):161-167, 2002

Nishinakamura, R., Matsumoto, Y., Nakao, K., Nakamura, K., Sato, A., Copeland, NG., Gilbert, DJ, Jenkins, NA., Scully, S., Lacey, DL., Katsuki, M., Asashima, M., and Yokota, T. Murine homolog of SALL1 is essential for ureteric bud invasion in kidney development . Development 128: 3105-3115, 2001

Nakayama, N., Han, C., Nishinakamura, R., Scully, S., He, C., Zeni, L., Yamane, H., Chang, D., Yu, D., Yokota, T., and Wen, D. A novel chordin-like protein inhibitor for bone morphogenetic proteins expressed preferentially in mesenchymal cell lineages. Dev. Biol. 232:372-387, 2001.

Sato, A., Asashima, M., Yokota, T. and Nishinakamura, R. Cloning and expression pattern of a Xenopus pronephros-specific gene, XSMP-30. Mech Dev. 92:273-275, 2000

Tomida, M., Heike, T., and Yokota, T. Cytoplasmic domains of the leukemia inhibitory factor receptor required for STAT3 activation, differentiation, andgrowth arrest of myeloid leukemic cells. Blood, 93: 1934-1941, 1999.

Matsuda, T., Nakamura, T., Nakao, K., Arai, T., Katsuki, M,. Heike, T. and Yokota, T. STAT3 activation is sufficient to maintain an undifferentiated state of mouse embryonic stem cells. EMBO J, 18:4261-4269, 1999.

Matsuzaki, K., Katayama, K., Takahashi, Y., Nakamura, I., Udagawa, N., Tsurukai, T., Nishinakamura, R., Toyama, Y., Yabe, Y., Hori, M., Takahashi, N., and Suda, T. Human osteoclast-like cells are formed from peripheral blood mononuclear cells in a coculture with SaOS-2 cells transfected with the parathyroid hormone (PTH)/PTH-related protein receptor gene. Endocrinology 140(2):925-932, 1999.

Takagi, M., Nakamura, T., Sawada, T., Kaneko, A., Nozaki-Ukai, M., Nakahata, T., Yokota, T. and Heike, T. Chimeric cytokine receptor can transduce expansion signals in interleukin 6 receptor alpha (IL-6Ralpha)-, IL-11Ralpha-,and gp130-low to -negative primitive hematopoietic progenitors. Mol Biol Cell. 10:3633-3642, 1999.

Onuma, Y., Nishinakamura, R., Takahashi, S., Yokota, T. and Asashima, M. Molecular cloning of a novel Xenopus spalt gene (Xsal-3). Biochem Biophys Res Commun. 264:151-156, 1999.

Nishinakamura, R., Matsumoto, Y., Matsuda, T., Ariizumi, T., Heike, T., Asashima, M. and Yokota, T. Activation of Stat3 by cytokine receptor gp130 ventralizes Xenopus embryos independent of BMP-4. Dev Biol. 216:481-490, 1999.

Nishinakamura, R., Matsumoto, Y., Uochi, T., Asashima, M., and Yokota, T. Xenopus FK 506-binding protein homolog induces a secondary axis in frog embryos, which is inhibited by coexisting BMP 4 signaling. Biochem. Biophys. Res. Commun. 239:585-591,1997.

Dirksen, U., Nishinakamura, R., Groneck, P., Hattenhorst, U., Nogee, L., Murray, R., and Burdach, S. Human pulmonary alveolar proteinosis associated with a defect in GM-CSF/IL-3/IL-5 receptor common βchain expression. J. Clin. Invest. 100:2211-2217, 1997.

Cooke, K.R., Nishinakamura, R., Martin, T.R., Kobzik, L., Brewer, J., Whitsett, J.A., Bungard, D., Murray, R., and Ferrara, J.L. Persistence of pulmonary pathology and abnormal lung function in IL-3/GM-CSF/IL-5 βc receptor-deficient mice despite correction of alveolar proteinosis after BMT. Bone Marrow Transplant. 20:657-662, 1997.

Mocci, S., Dalrymple, S.A., Nishinakamura, R., and Murray, R. The cytokine stew and innate resistance to L. monocytogenes. Immunol. Rev. 158:107-114, 1997.

Miyajima, A., Kinoshita, T., Wakao, H., Hara, T., Yoshimura, A., Nishinakamura, R., Murray, R., and Mui, A. Signal transduction by the GM-CSF, IL-3 and IL-5 receptors. Leukemia, Suppl. 3:418-422, 1997.

Nishinakamura, R., Wiler, R., Dirksen, U., Morikawa, Y., Arai, K., Miyajima, A., Burdach, S., and Murray, R. The pulmonary alveolar proteinosis in granulocyte macrophage colony-stimulating factor/ interleukins 3/5 βc receptor-deficient mice is reversed by bone marrow transplantation. J. Exp. Med. 183:2657-2662, 1996.

Nishinakamura, R., Miyajima, A., Mee, P.J., Tybulewicz, V.L.J., and Murray, R. Hematopoiesis in mice lacking the entire granulocyte-macrophage colony-stimulating factor/interleukin-3/interleukin-5 functions. Blood 88:2458-2464, 1996.

Nishinakamura, R., Burdach, S., Dirksen, U., and Murray, R. The in vivo role of the receptors for IL-3, GM-CSF, and IL-5 (βc andβIL3). Contemporary Immunology: Cytokine Knockouts. Humana Press Inc. 419-434, 1995.

Nishinakamura, R., Nakayama, N., Hirabayashi, Y., Inoue, T., Aud, D., McNeil, T., Azuma, S., Yoshida, S., Toyoda, Y., Arai, K., Miyajima, A., and Murry, R. Mice deficient for the IL-3/GM-CSF/IL-5 βc receptor exhibit lung pathology and impaired immune response, while βIL3 receptor-deficient mice are normal. Immunity 2:211-222, 1995.